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During the developing COVID-19 pandemic, the Nuffield Department of Medicine Research Building is taking measures to prevent the spread of the disease.

Departments are instructed by the University's Registrar to continue to work from home where possible, and manage the return to on site working, based on the University guidelines for risk assessments and work prioritisation.

This is to restrict contact between individuals as far as possible.  The University remains open and operating as far as possible with the following restrictions:

  • No public access to the University
  • On-site activity permitted where it cannot be undertaken remotely, driven by safety, capacity and other factors such as schools reopening/other changes in government guidelines
  • Teaching and assessment are undertaken remotely where possible and, depending on government guidelines, gatherings of staff and students only permitted where essential for teaching and assessment to take place

LATEST NEWS

Animation: Understanding COVID-19 transmission, informing control

7 Aug 2020 By Dr Fiona Jones, Digital Editor Oxford Sparks We’ve all heard of – and indeed been affected by – COVID-19, the disease caused by infection with the novel SARS-CoV-2 virus. We’ve also become familiar with a plethora of new terminology, with “social distancing”, “lockdown”, “flattening the curve” and “R number” regularly and effortlessly winding their way into our conversations. Something we might not be so familiar with, however, is the scientific process undertaken when we find ourselves faced with a new and unknown pathogen (whether that be a virus, bacterium or prion).

A novel strategy for using compounds as ‘anti-evolution’ drugs to combat antibiotic resistance

The rise of antibiotic resistance in many pathogens has been driven by the spread of a small number of strains, suggesting that some bacteria may be genetically pre-disposed to evolving resistance. Researchers at Oxford University have tested this hypothesis by quantifying differences in evolvability between pathogen strains and by searching for ‘potentiator’ genes that accelerate the evolution of resistance. Their results are published today in Nature Communications.

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